To the Editor: Scott and colleagues address the vexed issue of using highly sensitive (hs) cardiac troponin (cTn) assays to detect myocardial infarction (MI) and likely increases in hospitalisations.1 Greater numbers of MIs detected by hs-cTn assays also confound population surveillance of MI trends and our understanding of disease prevention.
The full article is accessible to AMA members and paid subscribers. Login to read more or purchase a subscription now.
Please note: institutional and Research4Life access to the MJA is now provided through Wiley Online Library.
- 1. Scott IA, Cullen L, Tate JR, Parsonage W. Highly sensitive troponin assays — a two-edged sword? Med J Aust 2012; 197: 320-323. <MJA full text>
- 2. Koukkunen H, Penttila K, Kemppainen A, et al. Differences in the diagnosis of myocardial infarction by troponin T compared with clinical and epidemiologic criteria. Am J Cardiol 2001; 88: 727-731.
- 3. Rosamond WD, Chambless LE, Heiss G, et al. Twenty-two year trends in incidence of myocardial infarction, coronary heart disease mortality, and case fatality in 4 US communities, 1987-2008. Circulation 2012; 125: 1848-1857.
- 4. Sanfilippo FM, Hobbs MST, Knuiman MW, et al. Can we monitor heart attack in the troponin era? Evidence from a population-based cohort study. BMC Cardiovasc Disord 2011; 11: 35.
- 5. Nedkoff L, Briffa T, Preen D, et al. Age- and sex-specific trends in the incidence of hospitalized acute coronary syndromes in Western Australia. Circ Cardiovasc Qual Outcomes 2011; 4: 557-564.
Online responses are no longer available. Please refer to our instructions for authors page for more information.
No relevant disclosures.